{
  "abstract": "Respiratory follow-up of children and adults born prematurely thus far has largely focused on those with the diagnostic label of bronchopulmonary dysplasia (BPD) which is assigned early in life.1 While BPD remains clinically important, especially during early infancy in those born extremely preterm (<28 weeks’ gestation), it has become increasingly apparent that it captures only a fraction of the respiratory burden experienced by individuals born preterm. Many preterm-born children, especially those born very (28–31 weeks’ gestation), moderate (32–33 weeks) and late (34–36 weeks) preterm, rarely develop BPD but often exhibit impaired lung function later in life, including in adult life,2 3 while a significant proportion who had BPD in infancy have relatively preserved respiratory outcomes. This disconnect has catalysed the development of the concept of prematurity-associated lung disease (PLD)4—a framework that encompasses the many risk factors beyond BPD and recognises the heterogeneous phenotypes5 and differing life-course trajectories6 rather than focusing on a single binary diagnosis.",
  "authors": [
    {
      "affiliations": [
        "Department of Child Health, Cardiff University, Cardiff, Wales, UK"
      ],
      "name": "Sailesh Kotecha"
    },
    {
      "affiliations": [
        "Department of Primary Care and Public Health, Cardiff University, Cardiff, Wales, UK"
      ],
      "name": "W John Watkins"
    }
  ],
  "title": "Beyond bronchopulmonary dysplasia: early-life determinants and divergent lung function trajectories of prematurity-associated lung disease",
  "uid": "3085bab5-0ae0-53ca-bf07-8105a12c1180"
}
