{
  "abstract": "Autoimmune glial fibrillary acidic protein (GFAP) astrocytopathy is a rare Central Nervous System (CNS) inflammatory disorder, first defined in 2016 and identified by GFAP-IgG in cerebrospinal fluid (CSF). Its rarity makes large-scale studies nearly impossible, so each well-designed case series is valuable. Within this context, Handa et al’s study is noteworthy for its design1; they included a well-matched disease control group and conducted thorough CSF analyses (Epstein-Barr virus (EBV) DNA PCR and serology), strengthening the validity of their findings.",
  "authors": [
    {
      "affiliations": [
        "Neuroinflammation, Queen Square Institute of Neurology, University College London, London, UK"
      ],
      "name": "Shannon A Bernard Healey"
    },
    {
      "affiliations": [
        "Neuroinflammation, Queen Square Institute of Neurology, University College London, London, UK"
      ],
      "name": "Laura Benjamin"
    }
  ],
  "title": "Epstein-Barr virus reactivation as a trigger in autoimmune GFAP astrocytopathy",
  "uid": "a532be42-4896-5b8b-9f1e-df93b9e27636"
}