{
  "abstract": "A man in his 20s presented to the emergency department with acute limb weakness. Laboratory investigations revealed severe hypokalaemia with serum potassium of 2.0 mmol/L. The 12-lead ECG at admission demonstrated distinctive ST-segment changes in the right precordial leads ( figure 1). Echocardiography showed no structural heart disease. Intravenous potassium supplementation was administered, promptly correcting the electrolyte disturbance to 4.2 mmol/L. A repeat ECG showed complete normalisation of the previously observed ST-segment abnormalities (figure 2). After full recovery, the patient underwent pharmacological provocation testing with intravenous flecainide (2 mg/kg over 10 min) in a monitored electrophysiology setting. The drug challenge reproducibly elicited an ECG pattern identical to that observed during the initial hypokalaemic episode (figure 3).",
  "authors": [
    {
      "affiliations": [
        "Jeonbuk National University Hospital, Jeonju-si, Jeollabuk-do, Korea (the Republic of)"
      ],
      "name": "Yisik Kim"
    },
    {
      "affiliations": [
        "Jeonbuk National University, Jeonju-si, Jeollabuk-do, Korea (the Republic of)"
      ],
      "name": "Kyung Pyo Kang"
    },
    {
      "affiliations": [
        "Jeonbuk National University Hospital, Jeonju-si, Jeollabuk-do, Korea (the Republic of)"
      ],
      "name": "Lae Young Jung"
    }
  ],
  "title": "Unmasking Brugada: when hypokalaemia reveals the hidden phenotype",
  "uid": "e6b47986-02bb-548d-a1b7-cc722fb5cb79"
}