{
  "abstract": "Liver fibrosis represents a serious worldwide health issue with a prevalence of around 5% in general populations.1 The most relevant aetiologies of liver fibrosis include metabolic dysfunction-associated steatotic liver disease (MASLD), alcohol-associated liver disease (ALD) and chronic hepatitis B and C infections. Persisting liver fibrosis can lead to irreversible liver cirrhosis with fatal complications such as ascites, variceal haemorrhage and acute-on-chronic liver failure.",
  "authors": [
    {
      "affiliations": [
        "Laboratori d’Oncologia Molecular, Bellvitge Biomedical Research Institute (IDIBELL), L'Hospitalet de Llobregat, Barcelona, Spain, Hospitalet de Llobregat, Barcelona, Spain"
      ],
      "name": "Isabel Fabregat"
    },
    {
      "affiliations": [
        "Dermatology, Heidelberg University Medical Faculty Mannheim, Mannheim, BW, Germany"
      ],
      "name": "Christian David Schmid"
    }
  ],
  "title": "Loss of sinusoidal endothelial identity: RAP1A at the crossroads of capillarisation and liver fibrosis",
  "uid": "134753cd-6b62-5884-aab7-fe5869a6850b"
}