{
  "abstract": "Kidney transplantation (KT) is the preferred treatment for end-stage renal disease, yet post-transplant complications and graft dysfunction remain significant challenges. Oxalate nephropathy, resulting from calcium oxalate crystal deposition due to hyperoxaluria, represents an under-recognised contributor to graft injury. We describe a woman in her 60s with obesity, chronic pancreatitis and type 2 diabetes who underwent KT after dialysis. Post-transplant, she developed recurrent pyelonephritis and impaired graft function. The biopsy revealed oxalate crystal deposits, tubular injury and interstitial nephritis and fibrosis consistent with oxalate nephropathy. Contributing factors included chronic pancreatitis-related malabsorption and high dietary vitamin C intake. Supportive measures, including a low-oxalate diet and hydration, were introduced. This case illustrates the multifactorial nature of allograft dysfunction and highlights the importance of considering oxalate nephropathy in at-risk patients. Early recognition and preventive strategies may help preserve graft function and improve long-term outcomes.",
  "authors": [
    {
      "affiliations": [
        "Nephrology Department, Unidade Local de Saúde de Almada-Seixal, Almada, Portugal"
      ],
      "name": "Filipa Fonte Rodrigues"
    },
    {
      "affiliations": [
        "Nephrology Department, Unidade Local de Saúde de Almada-Seixal, Almada, Portugal"
      ],
      "name": "Catarina Cardoso"
    },
    {
      "affiliations": [
        "Pathology Department, Unidade Local de Saúde de Amadora/Sintra, Amadora, Portugal"
      ],
      "name": "Rita Manso"
    },
    {
      "affiliations": [
        "Nephrology Department, Unidade Local de Saúde de Almada-Seixal, Almada, Portugal"
      ],
      "name": "Pedro Bravo"
    }
  ],
  "title": "Oxalate nephropathy in kidney transplant",
  "uid": "a5a5a086-63ed-5a05-8bcd-acd57f792799"
}